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Referencias:

• Harlen, K.M., Churchman, L.S., The code and beyond: transcription regulation by the RNA polymerase II carboxy-terminal domain (2017) Nat Rev Mol Cell Biol, 18, pp. 263-273
• Muñoz, M., de la Mata, M., Kornblihtt, A., The carboxy terminal domain of RNA polymerase II and alternative splicing (2010) Trends in biochemical sciences, 35, pp. 497-504
• Muñoz, M., Pérez Santangelo, M., Paronetto, M., DNA damage regulates alternative splicing through inhibition of RNA polymerase II elongation (2009) Cell, 137, pp. 708-720. , et al
• Nouspikel, T., DNA repair in mammalian cells: Nucleotide excision repair: variations on versatility (2009) Cellular and molecular life sciences: CMLS, 66, pp. 994-1009
• Anindya, R., Mari, P.-O., Kristensen, U., A ubiquitin-binding domain in Cockayne syndrome B required for transcription-coupled nucleotide excision repair (2010) Molecular cell, 38, pp. 637-648. , et al
• Marteijn, J.A., Lans, H., Vermeulen, W., Understanding nucleotide excision repair and its roles in cancer and ageing (2014) Nat Rev Mol Cell Biol, 15, pp. 465-481. , et al
• Compe, E., Egly, J.-M., TFIIH: when transcription met DNA repair (2012) Nat Rev Mol Cell Biol, 13, pp. 343-354
• Le May, M.-F.D., Velez-Cruz, R., Iltis, I., NER factors are recruited to active promoters and facilitate chromatin modification for transcription in the absence of exogenous genotoxic attack (2010) Mol Cell, 38, pp. 54-66. , et al
• Gyenis, A., Umlauf, D., Ujfaludi, Z., UVB induces a genome-wide acting negative regulatory mechanism that operates at the level of transcription initiation in human cells (2014) PLoS Genet, 10, p. e1004483. , et al
• Fong, Y.W., Cattoglio, C., Tjian, R., The intertwined roles of transcription and repair proteins (2013) Mol Cell, 52, pp. 291-302
• Hanawalt, P., Spivak, G., Transcription-coupled DNA repair: two decades of progress and surprises (2008) Nat Rev Mol Cell Biol, 9, pp. 958-970
• Mouret, S., Charveron, M., Favier, A., Differential repair of UVB-induced cyclobutane pyrimidine dimers in cultured human skin cells and whole human skin (2008) DNA Repair (Amst), 7, pp. 704-712. , et al
• D'Errico, M., Teson, M., Calcagnile, A., Differential role of transcription-coupled repair in UVB-induced response of human fibroblasts and keratinocytes (2005) Cancer Res, 65, pp. 432-438. , et al
• D'Errico, M., Lemma, T., Calcagnile, A., Cell type and DNA damage specific response of human skin cells to environmental agents (2007) Mutat Res, 614, pp. 37-47. , et al
• Munoz, M.J., Nieto Moreno, N., Giono, L.E., Major Roles for Pyrimidine Dimers, Nucleotide Excision Repair, and ATR in the Alternative Splicing Response to UV Irradiation (2017) Cell Rep, 18, pp. 2868-2879. , et al
• Tresini, M., Warmerdam, D.O., Kolovos, P., The core spliceosome as target and effector of non-canonical ATM signalling (2015) Nature, 523, pp. 53-58. , et al
• Hanasoge, S., Ljungman, M., H2AX phosphorylation after UV irradiation is triggered by DNA repair intermediates and is mediated by the ATR kinase (2007) Carcinogenesis, 28, pp. 2298-2304
• Marteijn, J.A., Bekker-Jensen, S., Mailand, N., Nucleotide excision repair-induced H2A ubiquitination is dependent on MDC1 and RNF8 and reveals a universal DNA damage response (2009) J Cell Biol, 186, pp. 835-847. , et al
• Matsumoto, M., Yaginuma, K., Igarashi, A., Perturbed gap-filling synthesis in nucleotide excision repair causes histone H2AX phosphorylation in human quiescent cells (2007) J Cell Sci, 120, pp. 1104-1112. , et al
• Stiff, T., Walker, S.A., Cerosaletti, K., ATR-dependent phosphorylation and activation of ATM in response to UV treatment or replication fork stalling (2006) EMBO J, 25, pp. 5775-5782. , et al
• Vrouwe, M.G., Pines, A., Overmeer, R.M., UV-induced photolesions elicit ATR-kinase-dependent signaling in non-cycling cells through nucleotide excision repair-dependent and -independent pathways (2011) J Cell Sci, 124, pp. 435-446. , et al
• Kumar, A., Mazzanti, M., Mistrik, M., ATR mediates a checkpoint at the nuclear envelope in response to mechanical stress (2014) Cell, 158, pp. 633-646. , et al
• Toledo, L.I., Murga, M., Gutierrez-Martinez, P., ATR signaling can drive cells into senescence in the absence of DNA breaks (2008) Genes Dev, 22, pp. 297-302. , et al
• Awasthi, P., Foiani, M., Kumar, A., ATM and ATR signaling at a glance (2015) J Cell Sci, 128, pp. 4255-4262
• Hilton, B.A., Li, Z., Musich, P.R., ATR Plays a Direct Antiapoptotic Role at Mitochondria, which Is Regulated by Prolyl Isomerase Pin1 (2016) Mol Cell, 61, p. 487. , et al
• Matsuoka, S., Ballif, B.A., Smogorzewska, A., ATM and ATR substrate analysis reveals extensive protein networks responsive to DNA damage (2007) Science, 316, pp. 1160-1166. , et al
• Mu, J.J., Wang, Y., Luo, H., A proteomic analysis of ataxia telangiectasia-mutated (ATM)/ATM-Rad3-related (ATR) substrates identifies the ubiquitin-proteasome system as a regulator for DNA damage checkpoints (2007) J Biol Chem, 282, pp. 17330-17334. , et al
• Paulsen, R.D., Soni, D.V., Wollman, R., A genome-wide siRNA screen reveals diverse cellular processes and pathways that mediate genome stability (2009) Mol Cell, 35, pp. 228-239. , et al
• Marini, F., Nardo, T., Giannattasio, M., DNA nucleotide excision repair-dependent signaling to checkpoint activation (2006) Proc Natl Acad Sci U S A, 103, pp. 17325-17330. , et al
• Godon, C., Mourgues, S., Nonnekens, J., Generation of DNA single-strand displacement by compromised nucleotide excision repair (2012) EMBO J, 31, pp. 3550-3563. , et al
• DiGiovanna, J.J., Kraemer, K.H., Shining a light on xeroderma pigmentosum (2012) J Invest Dermatol, 132, pp. 785-796
• Bukowska, B., Karwowski, B.T., Actual state of knowledge in the field of diseases related with defective nucleotide excision repair (2018) Life Sci, 195, pp. 6-18
• Bartkowiak, B., Liu, P., Phatnani, H.P., CDK12 is a transcription elongation-associated CTD kinase, the metazoan ortholog of yeast Ctk1 (2010) Genes Dev, 24, pp. 2303-2316. , et al

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