Registro:

Referencias:

• Lomen-Hoerth, C., Murphy, J., Langmore, S., Kramer, J.H., Olney, R.K., Miller, B., Are amyotrophic lateral sclerosis patients cognitively normal? (2003) Neurology, 60, pp. 1094-1097
• Strong, M.J., Lomen-Hoerth, C., Caselli, R.J., Bigio, E.H., Yang, W., Cognitive impairment, frontotemporal dementia, and the motor neuron diseases (2003) Ann Neurol, 54 (SUPPL. 5), pp. S20-S23
• Strong, M.J., The basic aspects of therapeutics in amyotrophic lateral sclerosis (2003) Pharmacol Ther, 98, pp. 379-414
• Ringholz, G.M., Appel, S.H., Bradshaw, M., Cooke, N.A., Mosnik, D.M., Schulz, P.E., Prevalence and patterns of cognitive impairment in sporadic ALS (2005) Neurology, 65, pp. 586-590
• Mitsuyama, Y., Presenile dementia with motor neuron disease (1993) Dementia, 4, pp. 137-142
• Mitsuyama, Y., Dementia with motor neuron disease (2000) Neuropathology, 20 (SUPPL.), pp. S79-S81
• De Carvalho, M., Chio, A., Dengler, R., Hecht, M., Weber, M., Swash, M., Neurophysiological measures in amyotrophic lateral sclerosis: Markers of progression in clinical trials (2005) Amyotroph Lateral Scler Other Motor Neuron Disord, 6, pp. 17-28
• Drachman, D.B., Fishman, P.S., Rothstein, J.D., Motomura, M., Lang, B., Vincent, A., Amyotrophic lateral sclerosis. An autoimmune disease? (1995) Adv Neurol, 68, pp. 59-65
• Field, E.J., Hughes, D., Toxicity of motor neurone disease serum for myelin in tissue culture (1965) Br Med J, 5475, pp. 1399-1401
• Roisen, F.J., Bartfeld, H., Donnenfeld, H., Baxter, J., Neuron specific in vitro cytotoxicity of sera from patients with amyotrophic lateral sclerosis (1982) Muscle Nerve, 5, pp. 48-53
• Smith, R.G., Alexianu, M.E., Crawford, G., Nyormoi, O., Stefani, E., Appel, S.H., Cytotoxicity of immunoglobulins from amyotrophic lateral sclerosis patients on a hybrid motoneuron cell line (1994) Proc Natl Acad Sci USA, 91, pp. 3393-3397
• Horwich, M.S., Engel, W.K., Chauvin, P.B., Amyotrophic lateral sclerosis sera applied to cultured motor neurons (1974) Arch Neurol, 30, pp. 332-333
• Liveson, J., Frey, H., Bornstein, M.B., The effect of serum from ALS patients on organotypic nerve and muscle tissue cultures (1975) Acta Neuropathol (Berl), 32, pp. 127-131
• Troost, D., Van den Oord, J.J., Vianney de Jong, J.M., Immunohistochemical characterization of the inflammatory infiltrate in amyotrophic lateral sclerosis (1990) Neuropathol Appl Neurobiol, 16, pp. 401-410
• Mulder, D.W., Lambert, E.H., Eaton, L.M., Myasthenic syndrome in patients with amyotrophic lateral sclerosis (1959) Neurology, 9, pp. 627-631
• Denys, E.H., Norris Jr., F.H., Amyotrophic lateral sclerosis. Impairment of neuromuscular transmission (1979) Arch Neurol, 36, pp. 202-205
• Maselli, R.A., Wollman, R.L., Leung, C., Distad, B., Palombi, S., Richman, D.P., Neuromuscular transmission in amyotrophic lateral sclerosis (1993) Muscle Nerve, 16, pp. 1193-1203
• Uchitel, O.D., Appel, S.H., Crawford, F., Sczcupak, L., Immunoglobulins from amyotrophic lateral sclerosis patients enhance spontaneous transmitter release from motornerve terminals (1988) Proc Natl Acad Sci USA, 85, pp. 7371-7374
• Uchitel, O.D., Scornik, F., Protti, D.A., Fumberg, C.G., Alvarez, V., Appel, S.H., Long-term neuromuscular dysfunction produced by passive transfer of amyotrophic lateral sclerosis immunoglobulins (1992) Neurology, 42, pp. 2175-2180
• Appel, S.H., Engelhardt, J.I., Garcia, J., Stefani, E., Autoimmunity and ALS: A comparison of animal models of immune-mediated motor neuron destruction and human ALS (1991) Adv Neurol, 56, pp. 405-412
• O'Shaughnessy, T.J., Yan, H., Kim, J., Middlekauff, E.H., Lee, K.W., Phillips, L.H., Amyotrophic lateral sclerosis: Serum factors enhance spontaneous and evoked transmitter release at the neuromuscular junction (1998) Muscle Nerve, 21, pp. 81-90
• Muchnik, S., Losavio, A., Lorenzo, S.D., Effect of amyotrophic lateral sclerosis serum on calcium channels related to spontaneous acetylcholine release (2002) Clin Neurophysiol, 113, pp. 1066-1071
• Mosier, D.R., Baldelli, P., Delbono, O., Smith, R.G., Alexianu, M.E., Appel, S.H., Amyotrophic lateral sclerosis immunoglobulins increase Ca2+ currents in a motoneuron cell line (1995) Ann Neurol, 37, pp. 102-109
• Yan, H.D., Lim, W., Lee, K.W., Kim, J., Sera from amyotrophic lateral sclerosis patients reduce high-voltage activated Ca2+ currents in mice dorsal root ganglion neurons (1997) Neurosci Lett, 235, pp. 69-72
• Carter, J.R., Mynlieff, M., Amyotrophic lateral sclerosis patient IgG alters voltage dependence of Ca2+ channels in dissociated rat motoneurons (2003) Neurosci Lett, 353, pp. 221-225
• Arsac, C., Raymond, C., Martin-Moutot, N., Dargent, B., Couraud, F., Pouget, J., Immunoassays fail to detect antibodies against neuronal calcium channels in amyotrophic lateral sclerosis serum (1996) Ann Neurol, 40, pp. 695-700
• Engelhardt, J.I., Siklos, L., Komuves, L., Smith, R.G., Appel, S.H., Antibodies to calcium channels from ALS patients passively transferred to mice selectively increase intracellular calcium and induce ultrastructural changes in motoneurons (1995) Synapse, 20, pp. 185-199
• Engelhardt, J.I., Siklos, L., Appel, S.H., Altered calcium homeostasis and ultrastructure in motoneurons of mice caused by passively transferred anti-motoneuronal IgG (1997) J Neuropathol Exp Neurol, 56, pp. 21-39
• Siklos, L., Engelhardt, J., Harati, Y., Smith, R.G., Joo, F., Appel, S.H., Ultrastructural evidence for altered calcium in motor nerve terminals in amyotropic lateral sclerosis (1996) Ann Neurol, 39, pp. 203-216
• Thomas, M.M., Dunn, S.M., Amyotrophic lateral sclerosis immunoglobulins are ineffective in altering calcium influx through presynaptic voltage-sensitive calcium channels (1997) Neurol Res, 19, pp. 129-134
• McGeer, E.G., McGeer, P.L., Pharmacologic approaches to the treatment of amyotrophic lateral sclerosis (2005) BioDrugs, 19, pp. 31-37
• Guegan, C., Vila, M., Rosoklija, G., Hays, A.P., Przedborski, S., Recruitment of the mitochondrial-dependent apoptotic pathway in amyotrophic lateral sclerosis (2001) J Neurosci, 21, pp. 6569-6576
• Brooks, B.R., Escorial, E., World Federation of Neurology criteria for the diagnosis of amyotrophic lateral sclerosis. Subcommittee on Motor Neuron Diseases/Amyotrophic Lateral Sclerosis of the World Federation of Neurology Research Group on Neuromuscular Diseases and the El Escorial "Clinical limits of amyotrophic lateral sclerosis" workshop contributors (1994) J Neurol Sci, 124 (SUPPL.), pp. 96-107
• Miyamoto, M.D., Binomial analysis of quantal transmitter release at glycerol treated frog neuromuscular junctions (1975) J Physiol, 250, pp. 121-142
• Katz, B., Miledi, R., The role of calcium in neuromuscular facilitation (1968) J Physiol, 195, pp. 481-492
• Magleby, K.L., Stevens, C.F., A quantitative description of end-plate currents (1972) J Physiol, 223, pp. 173-197
• Pagani, M.R., Reisin, R.C., Uchitel, O.D., Calcium signaling pathways mediating synaptic potentiation triggered by amyotrophic lateral sclerosis IgG in motor nerve terminals (2006) J Neurosci, 26, pp. 2661-2672
• Pagani, R., Song, M., McEnery, M., Qin, N., Tsien, R.W., Toro, L., Differential expression of alpha 1 and beta subunits of voltage dependent Ca2+ channel at the neuromuscular junction of normal and P/Q Ca2+ channel knockout mouse (2004) Neuroscience, 123, pp. 75-85
• Nyormoi, O., Proteolytic activity in amyotrophic lateral sclerosis IgG preparations (1996) Ann Neurol, 40, pp. 701-706
• Narita, K., Akita, T., Hachisuka, J., Huang, S., Ochi, K., Kuba, K., Functional coupling of Ca(2+) channels to ryanodine receptors at presynaptic terminals. Amplification of exocytosis and plasticity (2000) J Gen Physiol, 115, pp. 519-532
• Day, N.C., Wood, S.J., Ince, P.G., Volsen, S.G., Smith, W., Slater, C.R., Differential localization of voltage-dependent calcium channel alpha1 subunits at the human and rat neuromuscular junction (1997) J Neurosci, 17, pp. 6226-6235
• Kawamata, T., Akiyama, H., Yamada, T., McGeer, P.L., Immunologic reactions in amyotrophic lateral sclerosis brain and spinal cord tissue (1992) Am J Pathol, 140, pp. 691-707
• Smith, R.G., Hamilton, S., Hofmann, F., Schneider, T., Nastainczyk, W., Birnbaumer, L., Serum antibodies to L-type calcium channels in patients with amyotrophic lateral sclerosis (1992) N Engl J Med, 327, pp. 1721-1728
• Vincent, A., Immunology of the neuromuscular junction and presynaptic nerve terminal (1999) Curr Opin Neurol, 12, pp. 545-551
• Bullens, R.W., O'Hanlon, G.M., Wagner, E., Molenaar, P.C., Furukawa, K., Plomp, J.J., Complex gangliosides at the neuromuscular junction are membrane receptors for autoantibodies and botulinum neurotoxin but redundant for normal synaptic function (2002) J Neurosci, 22, pp. 6876-6884
• Athanassakis, I., Protopapadakis, E., Vassiliadis, S., Localization of pepstatin's inhibitory action during Fc-mediated antibody internalization: Possible implications for antibody-mediated viral transmission (2000) Cell Immunol, 199, pp. 81-88
• Ober, R.J., Radu, C.G., Ghetie, V., Ward, E.S., Differences in promiscuity for antibody-FcRn interactions across species: Implications for therapeutic antibodies (2001) Int Immunol, 13, pp. 1551-1559
• Medesan, C., Cianga, P., Mummert, M., Stanescu, D., Ghetie, V., Ward, E.S., Comparative studies of rat IgG to further delineate the Fc:FcRn interaction site (1998) Eur J Immunol, 28, pp. 2092-2100
• Berridge, M.J., Bootman, M.D., Roderick, H.L., Calcium signalling: Dynamics, homeostasis and remodelling (2003) Nat Rev Mol Cell Biol, 4, pp. 517-529
• Orrenius, S., Zhivotovsky, B., Nicotera, P., Regulation of cell death: The calcium-apoptosis link (2003) Nat Rev Mol Cell Biol, 4, pp. 552-565
• Jayaraman, T., Marks, A.R., T cells deficient in inositol 1,4,5-trisphosphate receptor are resistant to apoptosis (1997) Mol Cell Biol, 17, pp. 3005-3012
• Demestre, M., Pullen, A., Orrell, R.W., Orth, M., ALS-IgG-induced selective motor neurone apoptosis in rat mixed primary spinal cord cultures (2005) J Neurochem, 94, pp. 268-275
• Rizzuto, R., Brini, M., Murgia, M., Pozzan, T., Microdomains with high Ca2+ close to IP3-sensitive channels that are sensed by neighboring mitochondria (1993) Science, 262, pp. 744-747
• David, G., Barrett, E.F., Stimulation-evoked increases in cytosolic [Ca(2+)] in mouse motor nerve terminals are limited by mitochondrial uptake and are temperature-dependent (2000) J Neurosci, 20, pp. 7290-7296
• Fratantoni, S.A., Weisz, G., Pardal, A.M., Reisin, R.C., Uchitel, O.D., Amyotrophic lateral sclerosis IgG-treated neuromuscular junctions develop sensitivity to L-type calcium channel blocker (2000) Muscle Nerve, 23, pp. 543-550
• Pullen, A.H., Demestre, M., Howard, R.S., Orrell, R.W., Passive transfer of purified IgG from patients with amyotrophic lateral sclerosis to mice results in degeneration of motor neurons accompanied by Ca2+ enhancement (2004) Acta Neuropathol (Berl), 107, pp. 35-46
• Santafe, M.M., Urbano, F.J., Lanuza, M.A., Uchitel, O.D., Multiple types of calcium channels mediate transmitter release during functional recovery of botulinum toxin type A-poisoned mouse motor nerve terminals (2000) Neuroscience, 95, pp. 227-234
• Norris Jr., F.H., Gasteiger, E.L., Action potentials of single motor units in normal muscle (1955) Electroencephalogr Clin Neurophysiol Suppl, 7, pp. 115-125
• Dasgupta, A., Simpson, J.A., Relation between firing frequency of motor units and muscle tension in the human (1962) Electromyography, 2, pp. 117-128
• Brooks, B.R., Shodis, K.A., Lewis, D.H., Rawling, J.D., Sanjak, M., Belden, D.S., Natural history of amyotrophic lateral sclerosis. Quantification of symptoms, signs, strength, and function (1995) Adv Neurol, 68, pp. 163-184
• 6|. Mitchell JD. Amyotrophic lateral sclerosis: toxins and environment. Amyotroph Lateral Scler Other Motor Neuron Disord 2000; 1: 235-50; Chio, A., Benzi, G., Dossena, M., Mutani, R., Mora, G., Severely increased risk of amyotrophic lateral sclerosis among Italian professional football players (2005) Brain, 128, pp. 472-476
• Piazza, O., Siren, A.L., Ehrenreich, H., Soccer, neurotrauma and amyotrophic lateral sclerosis: Is there a connection? (2004) Curr Med Res Opin, 20, pp. 505-508
• Haley, R.W., Excess incidence of ALS in young Gulf War veterans (2003) Neurology, 61, pp. 750-756

Citas:

---------- APA ----------

---------- CHICAGO ----------

---------- MLA ----------

---------- VANCOUVER ----------